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Guide

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  1. LDN.png

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    "Naltrexone, sold under the brand names ReVia and Vivitrol among others, is a medication primarily used to manage alcohol or opioid dependence." It also "has been used in chronic pain such as fibromyalgia with new but well documented evidence of potential benefit." [1] There are anecdotal reports that it reduces flushing, neuropathic rosacea, neurogenic rosacea, and improves phenotype 1 LEARN MORE about Low Dose Naltrexone for rosacea

  2. isotretinoin.png.18e4a8242d7569d61fa2618

    Higher doses of isotretinoin, but more importantly, low dose isotretinoin (requires subscription), has been reported to be a successful treatment for rosacea. You should be aware of the risk/benefit ratio (requires subscriptionwith this treatment if you are not aware of these facts. Are you aware of isotretinoin induced rosacea (requires subscription)? LEARN MORE ABOUT TREATMENT FOR ISOTRETINOIN INDUCED ROSACEA (requires subscription).

  3. Accutane (Isotretinoin) has been used for many years to treat acne rosacea in higher doses so if you are taking high dose Accutane (20-40 mg or higher) (requires subscription to viewyou should read the list of anecdotal reports causing Accutane induced rosacea (requires subscription to view). If you are taking low dose Accutane (isotretinoin) 2.5 - 10 mg read this post (requires subscription to view), it would be pertinent for you to read this post .

     

    • The current, as of this date, most popular theory on rosacea was postulated by Dr. Richard Gallo at UCSD, who began postulating the 'innate immune system dysfunction' theory which is a complicated theory. It has become one of the most discussed and hopefully closer to the truth about what is at the heart of rosacea's cause, first postulated in an article in the June 1, 2004 Dermatolgy Times by Michelle Stephenson, who quotes Richard L. Gallo, M.D., Ph.D., saying rosacea may be an 'abnormality in the innate immune system...caused by too much cathelicidin." Dr Gallo says, "if we believe that the disease is caused by too much cathelicidin, we could develop a strategy to block the effects of the cathelicidins by making molecules that mimic that protein but don't have the same effects." LEARN MORERequires Subscription

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