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    All the board members have been diagnosed with rosacea and are volunteers who are available to answer your questions or listen to your concerns in the Member Forum for members and you will have to join to be able to post. One board member has a master of science biotechnology.

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    Johannes Schild [Director, Board Member]                                                                    
    Spanish Fort, Alabama, USA
    Schild Investments, LLC
    Marine Consultancy, LLC
    Johannes_Schild_Resumé.pdf
    contact

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    Pam Tobey [Secretary, Board Member]                                                                                       
    Arlington, Virginia, USA
    Retired, Washington Post, News Information Designer
    Visuals Director at the Beijing Review Magazine
    contact

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    Brady Barrows [Treasurer, Board Member]                                         
    Centre, Alabama, USA
    Founder
    About
    contact

     

     
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    Apurva Tathe [Board Member]                                                                            
    Bhilai, India
    Master of Science Biotechnology 
    contact

  • Posts

    • So how did you achieve remission?  What regimen treatment did you use?
    • Occupational Dermatoses Among Frontline Healthcare Workers During the COVID-19 Pandemic: A Cross-Sectional Survey. J Am Acad Dermatol. 2020 Sep 17;: Authors: Trepanowski N, Larson AR, Evers-Meltzer R PMID: 32950541 [PubMed - as supplied by publisher] {url} = URL to article Journal Pre-proof
    • Related Articles Paired Transcriptomic and Proteomic Analysis Implicates IL-1β in the Pathogenesis of Papulopustular Rosacea Explants. J Invest Dermatol. 2020 Sep 14;: Authors: Harden JL, Shih YH, Xu J, Li R, Rajendran D, Hofland H, Chang ALS Abstract Papulopustular rosacea (PPR) is a chronic inflammatory skin disease with limited treatment options. Although multiple pathways have been described to be upregulated in PPR, a mechanistic understanding of the key drivers and interaction between pathways in PPR pathology is lacking. In this study, we utilized PPR biopsy explants to integrate both differentially expressed genes (DEGs) and differently expressed proteins (DEPs) in paired non-lesional (NL) and lesional (LS) PPR tissue (n=5 patients). The results of this study identified 92 DEGs and 20 DEPs between paired PPR LS and NL explants. MAPK and TNF signaling pathways were the most significantly upregulated pathways in PPR LS tissue and aligned with DEPs identified in this study. Both MAPK and TNF signaling pathways highlighted IL-1β as a potential central mediator to PPR pathogenesis. In support of this, stimulation of NL explants with IL-1β resulted in a transcriptomic and proteomic profile similar to LS PPR. In this integrative transcriptomic and quantitative protein analysis, we identified several inflammatory genes, proteins, and pathways which may be contributing to PPR, as well as highlighted a potential role of IL-1β in driving inflammation in PPR. PMID: 32941918 [PubMed - as supplied by publisher] Full Text {url} = URL to article Interleukin 1 beta (IL-1β) is a cytokine.  Cytokines and Rosacea
    • Fluorescence-advanced videodermatoscopy characterization of demodex mites. A comparison with reflectance confocal microscopy. J Eur Acad Dermatol Venereol. 2020 Sep 14;: Authors: Fernandez-Nieto D, Segurado-Miravalles G, Gonzalez-Cantero A, Ortega-Quijano D, Jimenez-Cauhe J, Boixeda P Abstract Demodex mites (namely D. folliculorum and D. Brevis) are commonly considered commensals of human pilosebaceous units. However, they are thought to play a pathogenic role in several skin conditions, specially pityriasis folliculorum, rosacea, perioral dermatitis and hair folliculitis.1 Several studies report an increased density of demodex mites in patients with rosacea (papulopustular and erythematotelangiectatic forms. PMID: 32924177 [PubMed - as supplied by publisher] {url} = URL to article
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