Root Admin Guide Posted February 25, 2020 Root Admin Report Share Posted February 25, 2020 image courtesy of Wikimedia Commons There is a hypothesis that the 'cross reaction between the GroEl chaperonin antibodies against the B.oleronius and human GroEl chaperonin' 'will not fold normally the ALDH2, and then the enzyme will not metabolize the acetaldehyde' and therefore, 'As a result, high amounts of acetaldehyde will circulate for longer time in the blood, until the liver CYP2E1(p450) enzyme system finally metabilizes the acetaldehyde, during that period of time the patients will experience a flushing as well as the people with the "Asian flushing syndrome" suffer when they drink ethanol.' Med Hypotheses, 84 (4), 408-12 Apr 2015Hypothesis of Demodicidosis Rosacea Flushing Etiopathogenesis Mary Ann Robledo, Mariana Orduz Link to comment Share on other sites More sharing options...
Recommended Posts
Create an account or sign in to comment
You need to be a member in order to leave a comment
Create an account
Sign up for a new account in our community. It's easy!
Register a new accountSign in
Already have an account? Sign in here.
Sign In Now